Is there a risk of delayed Prion-type Neural Diseases from the Covid vaccines?

mRNA COVID-19 Vaccines and Risk of Prion Disease — Shocking Study

April 13, 2021 
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[Editor’s note: Gumshoe has written about prions before, but now we re-post a recent study — sourced from Greatgameindia and Medicdebate in an article entitled: Shocking Study Reveals mRNA COVID-19 Vaccines May Progressively Degenerate Your Brain From Prion Disease]

A study by David Sulzer, PhD, Columbia University Medical Center

April 10, 2021

A shocking study has revealed the terrifying dangers of mRNA COVID-19 vaccines inducing prion-based disease causing your brain to degenerate progressively. The mRNA vaccine induced prions may cause neurodegenerative diseases because long-term memories are maintained by prion-like proteins. The study concluded that mRNA based vaccine may also cause ALS, front temporal lobar degeneration, Alzheimer’s disease and other neurological degenerative diseases in the vaccine recipients.

Interaction between SARS-COV-2 Prions & ACE2 Receptors

The spike protein outer shell of the coronavirus contains “prion-like regions” that give the virus very high adhesion to ACE2 receptors in the human body.

This has been documented by a study entitled, “SARS-CoV-2 Prion-Like Domains in Spike Proteins Enable Higher Affinity to ACE2,” published by the Human Microbiology Institute:

Interactions between amino acids of PrDs and non-prion-like regions of SARS- CoV-2 RBD and ACE2.

The presence and unique distribution of prion-like domains in the SARS-CoV-2 receptor-binding domains of the spike protein is particularly interesting, since although the SARS-CoV-2 and SARS-CoV S proteins share the same host cell receptor, angiotensin-converting enzyme 2 (ACE2), SARS-CoV-2 demonstrates a 10- to 20-fold higher affinity for ACE2.

The Cross-Species Jump

This special relationship between the S protein and ACE2 receptor is the key to cross-species infection which made it possible for the coronovirus to make a jump from animals to humans.

The special relationship between the S protein and ACE2 receptor is the key to cross-species injection which made it possible for the coronovirus to make a jump from animals to humans.

A remarkable fact is that the Coronavirus carried by bats cannot directly infect the human body, let alone a virus that can possess human-to-human transmission.

This cross-species jump was not natural and was achieved by the team lead by the Batwoman of China, Shi Zhengli. You can read more about it in detail in COVID19 Files – Scientific Investigation On Mysterious Origin Of Coronavirus.

This is what Batwoman of China had to say when GreatGameIndia exposed the research done in Wuhan Institute of Virology.

It was funded under gain-of-function experiments through Peter Daszak, the President of EcoHealth Alliance by the US Government.

The Coverup

The interesting part is that Peter Daszak is the same guy who orchestrated the publication of a ‘scientific’ paper in Lancet claiming that the virus made the cross-species jump naturally. How can the same guy who funded the experiments to make the cross-species jump of the virus, claim that it evolved naturally?

If you think that’s outrageous, well just wait for it.

President of EcoHealth Alliance, Peter Daszak

Peter Daszak is also the same guy the WHO sent to China to investigate the claims whether the virus evolved naturally or was coronavirus engineered. Yet another person linked to the team is Dutch virologist Ron Fouchier, another key to the coronavirus investigation who also created the deadly mutant H5N1 virus.

We encourage our readers to explore more about the amazing personalities in the WHO investigative team.

COVID-19 mRNA Vaccines & Prion-based Diseases

What are Prions?

Because of such experiments by these eminent scientists the virus contains prions-like domains in its receptor region of the spike protein.

Prions are misfolded proteins with the ability to transmit their misfolded shape onto normal variants of the same protein. They characterize several fatal and transmissible neurodegenerative diseases in humans and many other animals.

How mRNA Vaccines Work

Messenger RNA (mRNA) is a single-stranded molecule naturally present in all of our cells. It carries the instructions for making proteins from our genes, located in the cell nucleus, to the cytoplasm, the main body of our cells. Enzymes in the cytoplasm then translate the information stored in mRNA and make proteins.

An mRNA vaccine delivers the instructions for making a bacterial or viral protein to our cells. Our immune system then responds to these proteins and develops the tools to react to future infections with the pathogen.

mRNA Vaccine-induced Prion-based diseases

Research has shown that RNA editing alterations cause prion diseases – fatal neurodegenerative disorders characterized by rapidly progressive dementia.

Research has shown that RNA editing alterations cause prion diseases – fatal neurodegenerative disorders characterized by rapidly progressive dementia.

Now when the mRNA vaccine triggers the immune response, the body may itself start creating prions induced by the vaccine mimicking its presence in the spike protein causing prion-based disease in the vaccine recipients.

The risk of this was assessed by Dr. J. Bart Classen, who authored a paper (read below) in Microbiology & Infectious Diseases titled “Covid-19 RNA Based Vaccines and the Risk of Prion Disease.”

Development of new vaccine technology has been plagued with problems in the past. The current RNA based SARS-CoV-2 vaccines were approved in the US using an emergency order without extensive long term safety testing.

The authors evaluated the vaccine for the potential to induce prion-based disease in vaccine recipients.

The intricate mechanisms of neurodegeneration in prion diseases

The RNA sequence of the vaccine, as well as the spike protein target interaction, were analyzed for the potential to convert intracellular RNA binding proteins TAR DNA binding protein (TDP-43) and Fused in Sarcoma (FUS) into their pathologic prion conformations. The results indicate that the vaccine RNA has specific sequences that may induce TDP-43 and FUS to fold into their pathologic prion confirmations.

The folding of TDP-43 and FUS into their pathologic prion confirmations is known to cause ALS, front temporal lobar degeneration, Alzheimer’s disease and other neurological degenerative diseases.

The finding of the study as well as additional potential risks lead the author to believe that regulatory approval of the RNA based vaccines for SARS-CoV-2 was premature and that the vaccine may cause much more harm than benefit.

Prions maintain Long-term Memories

The vaccine-induced prions may cause neurodegenerative diseases because long-term memories are maintained by prion-like proteins. The memory molecules are a normal version of prion proteins.

In four papers published in Neuron and Cell Reports, Dr. Kandel’s laboratory showed how prion-like proteins – similar to the prions behind mad cow disease in cattle and Creutzfeld-Jakob disease in humans – are critical for maintaining long-term memories in mice, and probably in other mammals.

When long-term memories are created in the brain, new connections are made between neurons to store the memory. But those physical connections must be maintained for a memory to persist, or else they will disintegrate and the memory will disappear within days.

Memories are stored for the long-term with the help of prion-like proteins called CPEB. CPEB prions aggregate and maintain synapses that recorded the memory.

When CPEB prions are not present or are inactivated, the synapses collapse and the memory fades.

– David Sulzer, PhD, Columbia University Medical Center

Extract from The Guardian of February 25, 2009

Obituary of Carleton Gajdusek (85)

Meanwhile, the Americans noted that the brains were similar to those of CJD patients. Burnet proposed sending out a multidisciplinary team. Gajdusek replied that he was that team.

Around this time he visited the Anga. They did not have kuru but did have an interesting form of welcome: the youths persistently offered to fellate him and regarded it as great fun.

After nine months, Gajdusek returned to NIH. There, an American scientist, William Hadlow, wrote saying how similar the brains looked to brains of sheep infected with scrapie.

Gajdusek inoculated chimps with extracts of Fore brains, knowing it would be a long time incubating, and went back to the hospital he had founded for the Fore.

He visited other tribes with paedophiliac traditions, and in 1963 brought to the US the first of his 56 adopted sons, a 12-year-old Anga boy, who landed barefoot in Washington with a bone through his nose. He put them all through high school, and many through university or medical school.

In 1965, two years after they had been inoculated, the chimps started to become ill. Gajdusek consulted a British expert on sheep scrapie, who confirmed that the chimps had died of the same disease that killed the Fore.

It was a triumphant moment for Gajdusek, says the science writer DT Max, author of The Family That Couldn’t Sleep, a history of prion disease research: proof that the disease was caused by an infectious agent. By 1976, when he received his Nobel prize, Gajdusek had published 150 papers.

He published a further 450 papers on “slow virus” diseases and ethnography. In 1974 an American neurologist and neuroscientist, Stanley Prusiner, entered the field and coined the term prion (for proteinaceous infectious particle, and incorporating the first two letters of his surname). Prusiner received a Nobel prize in 1997.